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1 - 10 of 19 results found for JAK1. Did you mean: jake

  1. Partial JAK1 deficiency is sufficient to impair intracellular control of mycobacterial...

    http://iris.ucl.ac.uk/iris/publication/1651386/1

  2. Biallelic JAK1 mutations in immunodeficient patient with mycobacterial infection

    http://iris.ucl.ac.uk/iris/publication/1199082/1

  3. JAK3 mutants transform hematopoietic cells through JAK1 activation, causing T-cell acute...

    http://iris.ucl.ac.uk/iris/publication/1694050/3

  4. Response to JAK1/2 Inhibition with Baricitinib in "Candle", "Savi"...

    http://iris.ucl.ac.uk/iris/publication/1504681/3

  5. Preliminary Response to JAK1/2 Inhibition with Baricitinib in...

    http://iris.ucl.ac.uk/iris/publication/1524612/3

  6. Issue 4 - January 2009 | UCL Institute of Cardiovascular Science -...

    17 human proteins associated with the growth hormone release pathway were annotated, including GH1, GHR, GHRH, GHRHR, HNF4A, IGF1, IL6, JAK1, JAK2, NR3C1, POU1F1, PRL, SOCS2, STAT3, STAT5A, STAT5B and TYK2.

    https://www.ucl.ac.uk/cardiovascular/cardiovascular/newsletters/accordion/2009/issue4

  7. Dr Siobhan Burns | Division of Infection and Immunity - UCL –...

    Biallelic JAK1 mutations in immunodeficient patient with mycobacterial infection.

    https://www.ucl.ac.uk/infection-immunity/people/dr-siobhan-burns

  8. Siobhan Burns | Institute of Immunity and Transplantation - UCL –...

    Biallelic JAK1 mutations in immunodeficient patient with mycobacterial infection.

    https://www.ucl.ac.uk/immunity-transplantation/people/siobhan-burns

  9. JAK1/2 inhibition with baricitinib in the treatment of autoinflammatory interferonopathies

    Patients with IFN-mediated autoinflammatory diseases, such as CANDLE and SAVI, present with a variety of severe manifestations, including fever and inflammatory organ damage, and have a high mortality rate. Many of these patients fail to respond to

    http://iris.ucl.ac.uk/iris/publication/1558601/1

  10. Exosomes from neuronal stem cells may protect the heart from ischaemia/reperfusion injury...

    Myocardial infarction requires urgent reperfusion to salvage viable heart tissue. However, reperfusion increases infarct size further by promoting mitochondrial damage in cardiomyocytes. Exosomes from a wide range of different cell sources have been

    http://iris.ucl.ac.uk/iris/publication/1856864/7

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