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  1. CD148 Deficiency in Fibroblasts Promotes the Development of Pulmonary Fibrosis.

    RATIONALE: The receptor-like protein tyrosine phosphatase eta (CD148/PTPRJ) exerts anti-fibrotic effects in experimental pulmonary fibrosis via interactions with its ligand syndecan-2; however, the role of CD148 in human pulmonary fibrosis remains

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